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Our understanding of the pathophysiologic mechanisms underlying PD continues to evolve

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To alter physiological H 2 S levels, we exposed heart tissue to ischaemia, which is predicted to increase the levels of H 2 S due to inhibition of its degradation by SQOR through the reduced CoQ pool 12,13

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Pro-inflammatory cytokines i.e., TNF- and IL-1 may activate the serotoninergic transporter (SERT) via the stimulation of the p38 MAPK pathway, increasing the concentration of other catecholamines in specific brain regions such as the anterior hypothalamus (Zhu et al